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Other molecular methods including next generation sequencing are utilized to evaluate for targetable mutations in NSCLC, including in thoracic small biopsy specimens

Building upon this theoretical framework, Jin et al (133) experimentally validated miR-214 as a direct target of lncKCNQ1OT1

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In atherosclerosis patients, repair of nuclear and mitochondrial DNA in vascular smooth muscle cells requires ATP and reactive oxygen species, leading to further mitochondrial dysfunction, accelerated plaque formation, and impaired smooth muscle cell function

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