glutathione borrelia Selection and characterization of DNA aptamers targeting the surface protein CspZ with high-throughput cross-over SELEX Utilizing Glutathione for Lyme Disease,

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However, p31-43 may be similar to TSN (Yao et al., 2019) in the mechanism of cell damage, both inhibiting Nrf2 activation by increasing Keap1 expression, resulting in a significantly lower Nrf2/Keap1 ratio than normal group cells, so that Nrf2 cannot effectively up-regulate the activity of antioxidant enzymes

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