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Results: RSV inhibited CD31 expression and capillarization of LSECs, and promoted LSECs to release NO by stimulating the activity of KLF2-eNOS signaling pathway, which suppressed the expression of SMA and collagen1 in HSCs, indicating that RSC blocked the activation of HSCs through the upregulation of KLF2-eNOS signaling in LSECs

A researcher who miscalculates concentration by a factor of two administers double the intended dose across an entire study, invalidating weeks of data collection

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This central mechanism of dysautonomia, partially driven by oxidative stress is likely an important one for the recovering critically ill patient who often has encountered a degree of autonomic dysfunction (311)