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Our results suggest that NADPH is a more sensitive biomarker for senescence-associated phenotype and elevating NADPH generation could be a compensatory mechanism to slow down EC senescence through suppressing oxidative stress 52

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Moreover, miR-25, a specific miRNA, can suppress the expression of microphthalmia-associated transcription factor (MITF), a crucial transcription factor in melanogenesis, thereby modulating the process of melanogenesis