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Concurrently, non-oxidized dopamine lowered intracellular Fe 2+ accumulation by approximately 3040% at 25 M, decreased MDA production by about 2535% at 25 M, and sustained GSH levels restored by roughly 2030% compared with the erastin group

Kiesel and Stan, 2017)

Importantly, cell viability was not affected, meaning the compound did not simply kill fat cells but changed their metabolic behavior

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The overactivation of ACSL4, LPCAT3, and LOXs produces a large amount of phospholipid peroxides, which trigger ferroptosis

PLA2G4A, a member of the cytoplasmic phospholipase family, catalyzes the hydrolysis of membrane phospholipids to release AA, which produces PGE2