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Mutations of FUS/TLS reduce interaction with U1-snRNP Association of FUS/TLS with U1-snRNP identified earlier by mass spectrometry (Fig

As with DPN, management targeting pathogenesis is not able to reverse established autonomic neuropathy

APAP can be synthesized in the body through O-dealkylation of the prodrug phenacetin, a pain-killer that was withdrawn from the market due to nephrotoxicity and carcinogenesis [3]

Meanwhile, the reference bradykinin receptor antagonist, Icatibant (HOE 140), demonstrated strong, dose-dependent inhibition, with 65.54 3.61% reduction in inflammation within the first hour (Fig

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