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Highlights 4 , 1126 (2013)

Mitochondrial dysfunction caused by oxidative stress is closely associated with the occurrence and development of neurodegenerative diseases [30, 31]

elegans presents different transcriptional responses depending on the fungal or bacterial infection origin (116, 140)

This decline accelerates during caloric restriction, making the deficit state particularly catabolic

-Enolase, in particular, was recurrently indicated as a novel autoantigen in systemic lupus erythematosus (SLE), systemic sclerosis (SSc) (Moscato et al., 2000