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Glutathione might inhibit the effects of cerebral infarction and enhance antiapoptotic signaling after ischemic stroke, suggesting that GSH may be a potent therapeutic antioxidant that can attenuate severe pathologies after ischemic stroke, and stimulating GSH synthesis through administration of GSH precursors and micronutrients like selenium can optimize GSH and GSH peroxidase for optimal antioxidant defense in cerebral ischemia (155, 156)

Chen W, Zhao H, Li Y

doi: 10.3389/fpls.2017.01061 54 HeileA

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Rock KD, Patisaul HB (2018) Environmental mechanisms of neurodevelopmental toxicity

The modulation of ion homeostasis by silicon in cadmium treated poplar callus cells