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doi: 10.1210/jc.2012-1616 72 PleiN.Babi LekoM.GunjaaI.ZemunikT

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et al: Melatonin suppresses ER stress-dependent proapoptotic effects via AMPK in bone mesenchymal stem cells during mitochondrial oxidative damage

In summary, contrary to traditional views that oxidative stress merely leads to increased DNA damage and disease progression, a deeper understanding of redox signaling reveals that redox modifications can enhance the activity of ATM, facilitating the formation of RAD51 foci